Candida albicans, a prominent species within the fungal realm of the human microbiota, habitually colonizes the gastrointestinal tract, reproductive system, skin, and oral cavity of the vast majority of individuals without eliciting symptomatic manifestations.
Generally perceived as an innocuous commensal organism, it should be noted that disturbances to host immune response, microbial shifts, or localized environmental disruptions could potentially provoke an overgrowth of C. albicans, precipitating infections. Such complications are particularly precarious for those individuals with compromised immune functionality or those with implanted medical devices. The pathogenic potential of C. albicans is primarily expressed via adhesion/invasion of host cells, enzymatic secretion, hyphal transformation, or biofilm formation.
Fig.1 Candida albicans biofilm formation.1
Reference
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